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Faculty & Staff Details

Photo of Lisa Brinn Name: Lisa Brinn
Position: Assistant Professor
Email: lisa.brinn@ttuhsc.edu
Bio Dr. Brinn is an Assistant Professor for the Department of Biomedical Sciences on the Abilene campus. She joined the Department in May 2007 as an Instructor. She received a B.S. (Biology) from Universidade Santa Ursula (Brazil) and both her M.S. (Morphology & Cell Biology) and Ph.D. (Psychobiology and Education) from Universidade de São Paulo (Brazil). Dr. Brinn completed a Post Doctorate Fellowship (Cell Biology/Physiology and Neurology) at the Washington University School of Medicine in the laboratories of Dr. Alan Pearlman, M.D., and Dr. Janice Brunstrom.

Education:

B.S. – Biology at Universidade Santa Ursula, Rio de Janeiro, RJ, Brazil. 1993
M.S. – Science at Universidade de São Paulo, Ribeirão Preto, SP, Brazil. 1996
PhD – Science at Universidade de São Paulo, Ribeirão Preto, SP, Brazil. 1999
Postdoctoral Fellowship – in brain development at Washington University School of Medicine, Saint Louis, MO, USA. 2006

Research Interests:

Understanding the molecular mechanisms involved during neocortical development. Layer formation in the developing cerebral cortex requires the movement of neurons from their site of origin to their final laminar position. The analysis of genetic mutations that lead to abnormal migration and layer formation in the developing cerebral cortex of mice and humans has led to important new discoveries regarding the molecular mechanisms that underlie these processes.

Publications:

Brinn, L. S. ; Leite, J. P.; Larson, R. E.; Martins, A. R. (2007) Differential patterns of myosin Va expression during the ontogenesis of the rat hippocampus. J. Comp. Neurol. (submitted).
Brinn, L. S. ; Martins, A. R.; Larson, R. E. (2007) Myosin Va participates in gliophilic neuronal migration, but not in neurophilic migration during rat corticogenesis (in preparation).
Brinn, L.S. ; Brunstrom, J.E. (2007) Selective prenatal and postnatal effects of Neurotrophin-4 on specific subclasses of interneurons in NT4-induced malformations of the rat neocortex. (in preparation).